Lipid metabolism pdf download

metabolism, including lipid metabolism, controls T cell differentiation, survival, and effector functions. Here, we report that development of T cell senescence driven by both malignant tumor cells and regulatory T cells is a general feature in cancers. Senescent T cells have active glucose metabolism but exhibit unbalanced lipid metab-olism.  · The proper functioning of intestinal lipid metabolism is important to guarantee adequate supply of energy (in the form of lipids) to the various organs in the body. Lipid metabolism during fasting Mobilization of lipid stores adipose tissue activation of HSL: TAG → glycerol + 3 NEFA albumin liver gluconeogenesis ketone bodies (for brain, muscles) (for brain) depletion of glycogen muscle proteins → AA TCA cycle intermediates acetylCoA excess (oxaloacetate) are used for gluconeogenesis.

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1 Lipid metabolism: Oxidation of saturated (-oxidation); Ketogenesis and ketolysis; biosynthesis of fatty acids, lipids; metabolism of cholesterol; Hormonal regulation of lipid metabolism. Lipid metabolism in the liver is a major source of ketone bodies (see Chapter 9) Clinical: Fatty acids (like glycogen) are a major energy source especially for muscle cells. Systemic primary carnitine deficiency—can't get long chain fatty acids into the mitochondria leading to accumulation. Lipid metabolism during fasting Mobilization of lipid stores adipose tissue activation of HSL: TAG → glycerol + 3 NEFA albumin liver gluconeogenesis ketone bodies (for brain, muscles) (for brain) depletion of glycogen muscle proteins → AA TCA cycle intermediates acetylCoA excess (oxaloacetate) are used for gluconeogenesis.